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Can CHK1 blockade help BRCA-wild-type resistant ovarian cancer?

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In 49 heavily pretreated BRCAwt platinum-resistant HGSOC patients, prexasertib yielded ~29–33% ORR with manageable but frequent neutropenia before early trial stop.

Source

The CHK1 inhibitor prexasertib in BRCA wild-type platinum-resistant recurrent high-grade serous ovarian carcinoma: a phase 2 trial

Giudice E, Huang TT, Nair JR, et al. · Nature communications · 2024

doi.org/10.1038/s41467-024-47215-6Read the full paper ↗41 citationscc by

Study at a glance

Design
Other — Open-label single-arm phase 2 basket cohorts (NCT02203513 cohorts 5–6) of IV prexasertib in BRCAwt platinum-resistant HGSOC; early sponsorship stop
N
N=49 · 24 biopsiable (cohort 5) + 25 non-biopsiable (cohort 6); 39 RECIST-evaluable
Population
Adults with BRCA wild-type platinum-resistant recurrent high-grade serous ovarian/related carcinoma
Outcome
ORR (primary), safety, PFS; exploratory replication-stress biomarkers

Structured fields used in claim comparison tables when every cited study has a complete layer.

What they did

An NCI open-label phase 2 enrolled cohorts with or without biopsiable disease, giving IV prexasertib 105 mg/m² every 2 weeks, assessing RECIST ORR, PFS, safety, and exploratory biopsy/blood biomarkers.

What they found

Among 39 evaluable patients, ORR was 33.3% (cohort 5) and 28.6% (cohort 6); median PFS 4 vs 6 months; neutropenia in 85.7%. High POLA1/POLE/GINS3 transcripts tracked with PFS <6 months.

The limits

What it doesn't show

Not a randomized comparative efficacy trial; early stop limited definitive primary-endpoint evaluation and generalizability.

Key terms

Prexasertib
CHK1 inhibitor tested as monotherapy in this phase 2 study.
BRCAwt HGSOC
High-grade serous ovarian carcinoma without BRCA1/2 mutation—majority of HGSOC.
Platinum-resistant
Recurrent disease with limited platinum benefit; high unmet need.
ORR / PFS
Objective response rate and progression-free survival endpoints.
Replication stress
DNA replication vulnerability that may sensitize tumors to ATR/CHK1 blockade.

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What clinical setting was studied?

Common questions

Were patients BRCA-mutated?

No—BRCA wild-type status was required.

Did it shrink tumors?

Yes in a minority—about one-third of evaluable patients responded by RECIST.

Main safety issue?

Cytopenias, especially neutropenia (~86%).

Why early stop?

Company withdrew investigational drug sponsorship/supply; primary endpoint not evaluable as planned.

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