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Should animal models copy the DSM?

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Validity criteria that ask animals to match whole DSM syndromes should be dropped in favour of well-understood symptoms or endophenotypes.

Source

Evaluating the validity of animal models of mental disorder: from modeling syndromes to modeling endophenotypes

van den Berg H · History and philosophy of the life sciences · 2022

doi.org/10.1007/s40656-022-00537-4Read the full paper ↗3 citationscc by

Study at a glance

Design
Other — Philosophy of psychiatry critique of Willner-style syndrome-matching validity for animal models
N
Conceptual analysis of validity criteria; not an empirical sample
Population
Animal models of mental disorder validated against DSM syndromes
Outcome
Replace whole-syndrome validity targets with symptoms or endophenotypes

Structured fields used in claim comparison tables when every cited study has a complete layer.

What they did

The author reconstructs how mid-century comparative psychiatrists such as McKinney and Bunney tried to model whole syndromes, how Willner’s predictive/face/construct validity packaged that ambition, and why heterogeneity in DSM depression makes one-to-one mapping fail.

What they found

Face validity as symptom similarity pushed researchers toward modelling entire syndromes. Two people can both meet DSM depression without sharing any symptoms. The author argues we should abandon the DSM as the validation standard and follow calls to model well-understood symptom clusters and endophenotypes.

The limits

What it doesn't show

This is not a new mouse experiment and does not prove any particular endophenotype is valid. The NIMH funding figures are cited as historical context, not as this paper’s own accounting.

Key terms

Face validity
Similarity of symptoms between the animal setup and the human condition.
Construct validity
Whether the model captures the theoretical mechanism of the human disorder.
Predictive validity
Whether treatments (e.g. drugs) that work in humans work in the model.
Endophenotype
A measurable component along the path from genes to disorder, narrower than a whole syndrome.

Flashcards

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Research intelligence for this paper

See its role on concept claims, tensions it is part of, placement history, and related discoveries.

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Quiz yourself

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Willner’s triad?

Common questions

What is Willner’s triad?

Predictive, face, and construct validity.

Why is DSM depression a bad target?

Two diagnosed people need not share any symptoms.

What instead?

Well-understood symptoms or endophenotypes, not whole manuals.

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