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Can gut microbes cause pregnancy complications?

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Two-sample MR of 18,340 MiBioGen participants versus FinnGen pregnancy GWAS found taxon-specific IVW signals—e.g. Deltaproteobacteria raising PPH risk (OR 1.26) and Butyricimonas lowering it (OR 0.79).

Source

Causal associations between gut microbiota and adverse pregnancy outcomes: A two-sample Mendelian randomization study

Li C, Liu C, Li N · Frontiers in microbiology · 2022

doi.org/10.3389/fmicb.2022.1059281Read the full paper ↗33 citationscc by

Study at a glance

Design
Mendelian randomisation — Two-sample MR of MiBioGen gut-microbiome GWAS vs FinnGen R5 adverse pregnancy outcomes, IVW primary with MR-Egger/MR-PRESSO
N
N=18340 · MiBioGen exposure GWAS: 18,340 participants, 24 cohorts, 196 taxa after dropping unnamed taxa; FinnGen R5 outcomes include PPH 3,670/98,626 and GDM 5,687/117,892
Population
Host-genetics–microbiome GWAS (MiBioGen) and Finnish pregnancy-complication GWAS (FinnGen R5)
Outcome
IVW odds ratios for postpartum hemorrhage, abruptio placentae, spontaneous abortion, PROM, GDM, preeclampsia/eclampsia, and preterm birth

Structured fields used in claim comparison tables when every cited study has a complete layer.

What they did

Used locus-wide SNPs (p < 1×10⁻⁵) from MiBioGen 16S/genotype data (18,340 people; 196 taxa) as instruments for six FinnGen R5 adverse pregnancy outcomes, with IVW as the main estimator plus MR-Egger, MR-PRESSO, Cochran Q, and leave-one-out.

What they found

Several genera showed IVW associations: Deltaproteobacteria with higher PPH (OR 1.26, 1.00–1.59), Butyricimonas with lower PPH (OR 0.79), and mixed AP/GDM signals including Holdemanella OR 0.41 for abruptio placentae. All F statistics were >10.

The limits

What it doesn't show

MR of 16S taxa is not a trial of probiotics; FinnGen endpoints are coded diagnoses, SNPs were not genome-wide significant (p < 1×10⁻⁵), and horizontal pleiotropy can remain even after MR-PRESSO.

Key terms

Two-sample MR
Causal estimate using genetic instruments for exposure from one GWAS and outcomes from another.
IVW
Inverse-variance weighted combination of SNP-specific Wald ratios; primary estimator here.
MiBioGen
Multi-ethnic host-genetics–gut microbiome GWAS (18,340 people; 24 cohorts).
FinnGen R5
Finnish biobank GWAS used for pregnancy-complication outcomes.
Instrumental variable
SNP assumed to affect the outcome only through the exposure taxon.
APOs
Adverse pregnancy outcomes such as PPH, GDM, PROM, and preeclampsia.

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Common questions

What was the exposure GWAS?

MiBioGen: 18,340 participants from 24 cohorts and 196 named taxa.

What was the main MR method?

Inverse-variance weighted (IVW) estimates.

Were instruments weak?

No—F statistics were all larger than 10.

Did Deltaproteobacteria raise PPH risk?

IVW OR 1.26 (1.00–1.59), p = 0.047.

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